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View Clinical Profile Biography Much of my clinical work is with general psychiatric inpatients, including persons with schizophrenia, bipolar disorder, depression and dementia. My outpatient work focuses primarily on persons with mood disorders.

My research focus has been to evaluate the neurological injury in patients who are on Extracorporeal life support, otherwise commonly referred to as Extracorporeal Membrane Oxygenation (ECMO). I have collaborated this work with biomedical engineers and scientists in the lab.

My research focus has been to evaluate the neurological injury in patients who are on Extracorporeal life support, otherwise commonly referred to as Extracorporeal Membrane Oxygenation (ECMO). My collaboration to study this started with Dr.

In graduate school, my research showed that altered mitochondrial metabolism driven by uncoupling proteins is sufficient to inhibit growth signaling and tumorigenesis. I also studied how cold exposure in mice leads to distinct temporal metabolism in various depots of white and brown adipose tissue.

My MD/MBA program helped set my interests not only in clinical medicine, but also in making healthcare systems perform better for everyone. Seeking new experiences, my wife and I moved to high-energy Chicago during residency, and then to beautiful Seattle for fellowship.

The overarching theme of my research studies has focused on understanding key genetic events that lead to cancer in an effort to identify novel targets that will help improve current therapies.

Since 2005, my lab has been using advanced genomics and bioinformatics approaches, including RNA-seq, ChIP-seq, GRO-seq, and ATAC-seq, and I have been instrumental in establishing these methodologies and building the related infrastructure at UT Southwestern.

Room# NA5-120F, Dallas, Texas, 75390-9148. email: kim.orth@utsouthwestern.edu Office Phone: 214-648-1685; FAX: 214-648-1488   Personal Statement My laboratory is very active in elucidating mechanisms used by virulence factors expressed by bacterial pathogens.

This discovery laid the groundwork for my studies investigating the organization of the circadian system and how an experimental protocol that approximates chronic jet-lag or shift-work disrupts the phase relationship among circadian clocks.

Cao J, Remaley AT, Guan W, Devaraj S, Tsai MY, Atherosclerosis 2021 06 327 1-4 Apolipoprotein B discordance with low-density lipoprotein cholesterol and non-high-density lipoprotein cholesterol in relation to coronary artery calcification in the Multi-Ethnic Study of Atherosclerosis (MESA).