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Researchers at UT Southwestern Medical Center studied obstructive sleep apnea in a large group of children and concluded that underweight children with this condition are more likely to have decreased height, tonsillar hypertrophy, and allergic rhinitis.

Lu Sun, Ph.D., Assistant Professor of Molecular Biology at UT Southwestern, has been awarded $1.5 million from the National Institutes of Health (NIH) to study interactions between neurons and glial cells in the brain, which could provide insight into the causes of neurological disorders.

A study of teens diagnosed with the vaping-linked respiratory disease EVALI revealed that most also had gastrointestinal symptoms and a history of psychosocial factors, including substance abuse.

A genome engineering technique developed at UT Southwestern Medical Center helped make the institution a research partner in a new $185 million National Institutes of Health (NIH) initiative to build on findings of the Human Genome Project.

Jere Mitchell, M.D., former director of the Harry S. Moss Heart Center at UT Southwestern Medical Center and an internationally recognized exercise physiologist whose seminal findings on maximal oxygen uptake changed conventional medical practice on bed rest and laid the foundation for central

Adenomyosis – an abnormal tissue growth into the muscular wall of the uterus that causes painful cramps and heavy or prolonged menstrual bleeding – is more common than generally appreciated

Jian Zhou, Ph.D., Assistant Professor in UT Southwestern’s Lyda Hill Department of Bioinformatics, has been awarded $1.5 million from the National Institutes of Health (NIH) to use artificial intelligence to investigate the three-dimensional structure of DNA and its impact on health.

A two-arm molecule can effectively deplete cancer-protecting cells inside tumors, allowing the immune system to fight off tumors without becoming overactive.

A study by UT Southwestern researchers finds that changing the biochemistry of parts of brain cells abolished the formation of amyloid beta plaques in a mouse model of Alzheimer’s disease.

Using a virus to purposely mutate genes that produce cancer-driving proteins could shed light on the resistance that inevitably develops to cancer drugs that target them, a new study led by UT Southwestern scientists suggests.