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Differential splicing and glycosylation of Apoer2 alters synaptic plasticity and fear learning Catherine R. Wasser1,2,*,>†, Irene Masiulis2,†, Murat S. Durakoglugil1,2, Courtney Lane- Donovan1,2, Xunde Xian1,2, Uwe Beffert2, Anandita Agarwala2, Robert E. Hammer3, and Joachim Herz1,2,4,5,* 1Center for Translational Neurodegeneration Research, University of Texas Southwestern Medical Center, Dallas, TX 75390, USA 2Department of Molecular Genetics, University of Texas Southwestern Medical

1Scientific RePorTS | (2018) 8:4501 | DOI:10.1038/s41598-018-22819-3 www.nature.com/scientificreports Loss of the adaptor protein ShcA in endothelial cells protects against monocyte macrophage adhesion, LDL-oxydation, and atherosclerotic lesion formation Antoine Abou-Jaoude1, Lise Badiqué1, Mohamed Mlih1, Sara Awan1, Sunning Guo1, Alexandre Lemle1, Clauda Abboud1, Sophie Foppolo1, Lionel Host1, Jérôme Terrand1, Hélène Justiniano1, Joachim Herz2, Rachel L. Matz1 & Philippe Boucher   1

Loss of Reelin protects against atherosclerosis by reducing leukocyte-endothelial adhesion and lesion macrophage accumulation Yinyuan Ding1,2,3,*, Linzhang Huang4,*, Xunde Xian1,2,*, Ivan S. Yuhanna4, Catherine R. Wasser1,2, Michael Frotscher5, Chieko Mineo4, Philip W. Shaul4,†, and Joachim Herz1,2,6,7,8,† 1Department of Molecular Genetics, UT Southwestern Medical Center, Dallas, TX 75390, USA. 2Center for Translational Neurodegeneration Research, UT Southwestern Medical Center, Dallas

1 updated May 2025 Sendai virus reprogrammed iPSC Lines * Clone used for CRISPR/Cas9 editing of GAA repeats Line Clone # Sex GAA repeat number in fibroblasts GAA repeat number in iPSCs Karyotype data Karyotype passage CTRL 3348 6 M Control size Control size Yes, normal p11 CTRL 7522 5 F Control size Control size Yes, normal p15 CTRL 7522 12 F Control size Control size Yes, normal p13 CTRL 6718 1 M Control size Control size Yes, normal p4 CTRL 6718 6 M

Postdoctoral Training in Signaling and Gene Regulation using Proteomics and Mass Spectroscopy Dr. W. Lee Kraus is currently seeking applicants with a Ph.D. degree and significant experience in mass spectroscopy for postdoctoral training in the Laboratory of Signaling and Gene Regulation within the Cecil H. and Ida Green Center for Reproductive Biology Sciences. The research in the Kraus lab covers a broad array of topics, including signaling, gene regulation, and genome function

Structural Biology and Biochemistry of Gene Regulation A joint postdoctoral training position is available in the laboratories of Dr. W. Lee Kraus and Dr. Xin Liu in the Cecil H. and Ida Green Center for Reproductive Biology Sciences at UT Southwestern Medical Center to study signaling and gene regulation and molecular biophysics of gene regulation. We are seeking a trainee interested in studying gene regulatory complexes, who will initiate and execute research projects

Postdoctoral Training in Gene Regulation Molecular Mechanisms, Computational Genomics, Mouse Genetic Models, RNA Biology and Noncoding RNAs, Protein Structure, Translational/Clinical A postdoctoral training position is available in the laboratory of Dr. W. Lee Kraus, in the Cecil H. and Ida Green Center for Reproductive Biology Sciences at UT Southwestern Medical Center to study signaling and gene regulation. The Kraus Lab has several exciting projects related to a broad array of

Postdoctoral Training in Bioinformatics and Computational Biology Studies on the Genomics of Gene Regulation A postdoctoral training position is available in the laboratory of Dr. W. Lee Kraus, in the Cecil H. and Ida Green Center for Reproductive Biology Sciences at UT Southwestern Medical Center to study gene regulation on a global scale. The Kraus lab has many exciting, ongoing projects related to the genomics of signal-regulated gene expression in various biological models

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ARTICLE Received 23 May 2016 | Accepted 13 Sep 2016 | Published 27 Oct 2016 Molecular basis of cooperativity in pH-triggered supramolecular self-assembly Yang Li1, Tian Zhao1, Chensu Wang1,2, Zhiqiang Lin1, Gang Huang1, Baran D. Sumer3 & Jinming Gao1 Supramolecular self-assembly offers a powerful strategy to produce high-performance, stimuli-responsive nanomaterials. However, lack of molecular understanding of stimulated responses frequently hampers our ability to rationally design