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The primary goal of Henkemeyer laboratory is to understand the biochemical signals that regulate cell-cell interactions during embryonic development. 

Skip to main content Publications for Qing Zou, Ph.D.

Skip to main content Publications Lafora disease gene therapy: EPM2A but not EPM2B overexpression results in Lafora body formation Alao EO, Sheibani M, Wu J, Marriam U, Evans D, Kasiri S, Verma M, Nitschke S, Nitschke F, Gray SJ, Mitra S, Minassian BA 2026 Mar Neurotherapeutics 23 CUL1 variants cause…

Minassian Lab has been involved in the identification and co-discovery of the causative gene mutations in over 20 different childhood neurological diseases. 

Our lab seeks to uncover the structure-function relationship of macromolecules involved in protein misfolding — a key element of Alzheimer’s and other neurodegenerative diseases.

Michaely Lab focuses on the function of the proteins that control plasma membrane function. We have on-going projects investigating ARH/LDLR endocytosis and caveolae signal transduction. 

The general focus of the Green Lab is to understand the molecular mechanism of the mammalian circadian clock, how it controls rhythmic biochemistry, physiology and behavior and how loss of clock function can impact health, resulting in metabolic disease, cancer and other ailments.

Skip to main content Lab Members Current Lab Members Jongwook Park, Ph.D. Postdoctoral Research Fellow Dr. Jongwook Park received his Ph.D. from Korea University, where he studied the mechanisms of antibiotic resistance in pathogens and microbiota-mediated maternal effects. He joined the Obata Lab in…

Our lab are studying host-microbe interactions and tissue immunity using multiple model organisms i) Identifying novel host factors involved in bacterial and fungal sensing to regulate inflammatory diseases. ii) Identifying new microbial factors involved in bacterial and fungal sensing to regulate inflammatory diseases. These factors may include pore-forming toxins and other toxins. iii) Identifying how microbe sensing of host-derived factors in the gut controls microbial commensalism and pathogenicity. iv) Utilizing fungi as a model system, genetic engineering of microbial effectors to understand how a microbe dictates protective immunity, and pathogenic immunity at tissue barriers.

Learn more about the Wise-Rios Lab.