2017 Article Archive
UT Southwestern Simmons Cancer Center researchers have shown that a first-in-class molecule can prevent breast cancer growth when traditional therapies stop working.
UT Southwestern Medical Center scientists have identified a key protein that helps trigger ketamine’s rapid antidepressant effects in the brain, a crucial step to developing alternative treatments to the controversial drug being dispensed in a growing number of clinics across the country.
UT Southwestern Medical Center research supports an evidence-based medicine (EBM) approach that embraces individualized care to prevent overtreatment, specifically for patients with type 2 diabetes.
Dr. Elizabeth M. McNally, Director of the Center for Genetic Medicine at Northwestern University Feinberg School of Medicine, and holder of the 19th Ida M. Green Distinguished Visiting Professorship, Honoring Women in Science & Medicine, will deliver the lecture named to honor late Mrs. Green
The 2017 commemoration at UT Southwestern Medical Center of the legacy of Dr. Martin Luther King, Jr. highlighted the importance of service to the community, diversity in science and medicine, and promoting justice in various aspects of society, including health care.
Hillary Evans, a second-year Medical School student and an Albert Schweitzer Foundation Fellow, was presented the 2017 Martin Luther King Scholarship for Community Service in the recent MLK Day Commemoration at UT Southwestern Medical Center.
Speaking to UT Southwestern Medical School students, Dr. Marshall Wolf, Professor of Medicine at Harvard Medical School’s Brigham and Women’s Hospital, recently offered advice, counsel, and tips on becoming a better physician.
Researchers at the Peter O’Donnell Jr. Brain Institute at UT Southwestern Medical Center have compiled a review outlining how a specific form of protein increases the risk of developing Alzheimer’s disease.
A molecule that helps neurons communicate also controls how quickly brain cells reform neurotransmitter-storing packets needed to continue messaging with other cells, according to a new study from researchers at the Peter O’Donnell Jr. Brain Institute at UT Southwestern Medical Center.
The finding could explain why patients with BRCA1 mutations do not have an elevated risk for leukemia. The stem cells die before they have an opportunity to transform into a blood cancer.